Library
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Bromethalin is a common rodenticide. Cats are commonly poisoned when they find loose bait placed for rodent control. Relay toxicity, where poisoning occurs due to ingesting rodents that died from bromethalin, has been anecdotally reported in cats. Clinical signs can develop as soon as two hours after exposure and as long as 24-48 hours after exposure. There is no antidote for bromethalin, so prompt induction of vomiting by a veterinarian can be lifesaving. Prognosis is poor to grave if severe clinical signs develop such as seizures, paralysis, or coma. Patients with milder clinical signs may recover after several weeks but may have permanent neurologic damage.
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Cholecalciferol (vitamin D3) has become increasingly more common as a residential rodenticide. While it may seem like a safer option than anticoagulants, there is no antidote available and the amount in a single block is a severe overdose in nearly all animals. This handout discusses clinical signs, treatments, and prognosis for cholecalciferol poisoning in pets.
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Bromethalin is a common rodenticide. Dogs are commonly poisoned when they find loose blocks placed for rodent control, or they access the bulk container. Clinical signs can develop as soon as 2 hours after exposure and as long as 5 days after exposure. There is no antidote for bromethalin, so promptly inducing vomiting can be lifesaving. Prognosis is poor to grave if severe clinical signs develop such as seizures, paralysis, or coma. Patients with milder clinical signs may recover after several weeks but may have permanent neurologic damage.
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Vitamin D poisoning occurs when a dog ingests a toxic dose of vitamin D. Common sources of vitamin D poisoning include rodenticides that contain vitamin D and certain human medications.
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Vitamin D poisoning occurs when a dog ingests a toxic dose of vitamin D. Common sources of vitamin D poisoning include rodenticides that contain vitamin D and certain human medications.
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Chlorfenapyr poisoning is suspected if a pet has access to this pesticide and subsequently develops expected signs; it should also be suspected in a pet exhibiting signs and at risk of malicious poisoning. Pets that develop clinical signs following chlorfenapyr ingestion are unlikely to survive and chronic complications may be present for survivors.
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Aspirin is a commonly used, over-the-counter, nonsteroidal anti-inflammatory drug (NSAID) and is used to treat fever, pain, swelling, and clotting disorders in humans. Aspirin poisoning occurs when a cat ingests a toxic dose of aspirin, either through misuse or accidentally. Cats are especially sensitive to aspirin and salicylates, and as a result are more vulnerable to poisoning than dogs. High doses may damage major organs like the liver, kidneys, and central nervous system.
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Arsenic poisoning is the accidental ingestion, skin contact, or inhalation of products containing a toxic dose of arsenic. Clinical signs can include abdominal pain, salivation, vomiting, diarrhea, staggering, weakness, rapid weak pulse, lethargy, low body temperature, collapse, and death. Supportive therapy is a crucial part of treating arsenic poisoning.
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Arsenic poisoning is the accidental ingestion, skin contact, or inhalation of products containing a toxic dose of arsenic. Clinical signs can include abdominal pain, salivation, vomiting, diarrhea, staggering, weakness, rapid weak pulse, lethargy, low body temperature, collapse, and death. Supportive therapy is a crucial part of treating arsenic poisoning.
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Ethanol overdose by ingestion, inhalation, or skin absorption leads to alcohol poisoning. Isopropanol (isopropyl alcohol/rubbing alcohol) and methanol (methyl alcohol) can also cause alcohol poisoning. If a pet is not showing clinical signs, decontamination may be performed, including inducing vomiting (for ingestions) or bathing (for skin exposure). Other therapies are discussed. Recovery is expected within 24–36 hours of the onset of signs.
